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1.The results are: ρm= 3.57 × 103kg/m3, α = 204 dyne/cm at 1270℃, and ρm = 3.42 103kg/m3, α = 192 dyne/cm at 1300℃.
在1300℃时ρ_m=3.42×10~3kg/m~3,α=192dyne/cm。收藏指正
2.There was no significant difference in the ratio of TXB2/6-k eto-PGF1α between group A(△W>3.0kg)and group B(△W<3.0kg).
透析间期体重增加＞3．0kg与＜3．0kg者的血浆TXB2、6－keto－PGF1α水平及TXB2／6－keto－PGF1α比值无显著性差异。收藏指正
3.The basal dietary vitamin E concentration was 13mg/kg,and the five different α tocopherol acetate supplementation levels were 0,5,10,50 and 100mg/kg respectively.
各组的基础日粮相同，含α?生育酚13mg／kg，各组日粮中分别添加dl?α?生育酚醋酸酯0、5、10、50和100mg／kg。收藏指正
4.LPS (E coli O 111 B 4, 5 mg/kg) was injected intravenously to Wistar rats, and was immediately followed by iv injection of Dex 5 mg/kg, Cyp 5 mg/kg or Ani 10 mg/kg. The livers were taken at 2h after LPS challenga. TNFα mRNA levels were assessed by Northern blot.
Wistar大鼠40 只， 静脉注射LPS（EcoliO111B4 5m g／kg） 后， 立即静脉给予Dex 5m g／kg、Cyp5m g／kg 或Am i10m g／kg，于LPS攻击后2h 取动物的肝脏，APAAP法进行TNFα免疫组织化学研究，North－ern 杂交分析TNFαm RNA 表达水平。收藏指正
5.In this paper, whether NE and related drugs produce a similar action on the motility of small intestine was examined by the transportation of Indian ink in the intestinal tract. Subcutaneous injections of NE (4 mg·kg???1?),isoprenaline (Iso,10 mg·kg???1?) and xylazine (1.5 mg·kg???1?) inhibited the passage of ink significantly.
结果：α、β受体激动剂NE4mg／kg，β受体激动剂异丙肾上腺素（ISO）10mg／kg和α2受体激动剂噻拉嗪1．5mg／kg均抑制小肠运动；收藏指正
6.To study which ions in CaCO 3 was responsible for the accumulation of α-KG, the effects of different pH buffers on the α-KG accumulation were studied. The level of α-KG was found to correlate with the levels of both Ca 2+ and CO 3 2-, with Ca 2+ played a dominant role and CO 3 2- played a minor role.
用不同物质调节发酵液中pH的实验证实 :在丙酮酸发酵过程中 ,Ca2 + 对α KG的积累起主要作用 ,CO32 - 起辅助作用 ,两者对α KG的积累具有协同效应。收藏指正
7.Methods: One hundred and twenty Wistar rats were randomizedly divided into 5 groups, including sham IR group (SIR), IR group (IR), treated with 0.5mg/kg α-MSH after IR (SMSH), treated with 2.5mg/kg α-MSH after IR (LMSH) and treated with 2mg/kg dexamethason after IR (DEX).
方法:实验分5组,即假手术组(SIR)、缺血再灌注组(IR)、小剂量(0.5mg/kg)α-MSH干预组(SMSH)、大剂量(2.5mg/kg)α-MSH干预组(LMSH)和地塞米松(2mg/kg)干预组(DEX)。收藏指正
8.IHIFN α hasn′t synergistic or additive effect to cy 15 mg/kg or 5 Fu 10 mg/kg in the index of inhibitory rate in S 180 mice and survival days in EAC cells;
IHIFN－α与5－Fu10mg／kg合用对S180小鼠抑瘤率以及EAC小鼠存活天数并无增效作用；收藏指正
9.2. The acute test results showed that the LD50 of α-TOS in mice and rats were 1000mg/kg·bw and over.
应用HPLC 检测证明,α-TOS 诱导胃癌SGC-7901 细胞凋亡是以不分解的整体分子形式发挥作用的。收藏指正
10.The protective effects of Agm could be completely abolished by idazoxan (2 mg/kg), an α 2 adrenoceptor (α 2 AR) and imidazoline receptor (IR) antagonist, and be partially blocked by yohimbine (1 mg/kg), a selective α 2 AR antagonist.
应用咪唑啉受体和α2 肾上腺素能受体 (α2 adrenoceptor ,α2 AR)拮抗剂咪唑克生 (2mg/kg)可完全消除AGM的心肌保护作用。 应用α2 AR阻断剂育亨宾 (1mg/kg)能部分阻断AGM的上述作用。收藏指正
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