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1.The results showed that the averaged open time and NPo were markedly increased by Bay K8644, but had no effect on channel conductance.
结果显示 ,在BayK8644作用下 ,通道开放形式发生变化 ,明显可见多级开放 ; 通道平均开放时间、平均开放概率显著增加 ,但单通道电导无明显变化。收藏指正
2.(3) Pretreatment with either L-type calcium channel agonist Bay K8644 (0.25 μmol/L) or elevation of Ca2+ concentration (5 mmol/L) in superfusate antagonized the effects of SNP (10-4 mol/L) on AV node cells.
(3)应用L型钙通道开放剂Bay K8644(0.25μmol/L)或者提高灌流液中钙离子浓度(5 mmol/L)均可增加动作电位的APA，Vmax，VDD和RSF。 加入SNP（10-4 mol/L）可完全逆转Bay K8644和高Ca2+对房室结细胞的兴奋效应；收藏指正
3.② Pretreatment with proglumide (100 μmol·L -1), a nonselective CCK receptor antagonist, or Bay K8644 (0.5 μmol·L -1), an agonist of calcium channel, partially attenuated the inhibitory effect of CCK-8 (0.5 μmol·L -1) on CSB activity.
②预先灌流CCK 8非特异性受体阻断剂丙谷胺 10 0 μmol·L- 1或钙通道开放剂BayK86 440 .5 μmol·L- 1可部分阻断CCK 80 .5 μmol·L- 1对颈动脉窦压力感受器放电活动的抑制作用。 预先灌流一氧化氮合酶抑制剂L NAME不能阻断CCK 8的效应。收藏指正
4.RESULTS Ca 2+ currents were obtained by stepping membrane potential to voltage between -80 and +10 mV, in 10 mV increments from a holding potential of -90 mV. Accordingly, the inhibitory percentage of NiCl 2 on Ca 2+ current was more than that of CdCl 2. Furthermore, Bay K8644, an agonist of L type Ca 2+ channel, had little effects on these Ca 2+ currents.
结果阻断K+ 电流后 ,当钳制电位 -90mV、指令电压 -80～ +1 0mV、步阶电压 1 0mV时 ,记录到Ca2 +电流 ,二价无机阳离子对Ca2 + 电流的抑制作用Ni2 +>Cd2 + ,而L型Ca2 + 通道激动剂BayK8644对电流无任何影响。收藏指正
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