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1.CCK Receptor and Anti-inflammatory Intracellular Signaling Mechanisms Activated by CCK-8
CCK-8抗炎作用的受体及信号转导机制的研究收藏指正
2.1 CCK-8 attenuating FCI/R injury in rats
icv注射CCK-8后动脉血压仅出现轻微波动，10 min-15 min内恢复至给药前水平。收藏指正
3.Regulatory Effect of CCK-8 on LPS-induced NOS in Vascular Endothelial Cells
CCK-8对脂多糖诱导血管内皮细胞一氧化氮合酶表达的调节作用收藏指正
4.Changes of plasma CCK-8,VIP,SS,GAS during acute stage of trauma
创伤急性期血浆内CCK-8,VIP,SS,GAS含量变化收藏指正
5.METHODS: Eighty-four Sprague-Dawley rats were randomly divided into seven groups: control,LPS(instilled intratracheally to reproduce the model of ALI),NaHS(H2S donor) +LPS,propargylglycine [inhibitor of cysathionine-γ-lyase(CSE),PPG]+LPS,CCK-8+LPS,PPG+CCK-8+LPS and CCK-8 group.
方法:将84只SD大鼠随机分为正常对照组、LPS组(经气管内滴注LPS复制ALI)、NaHS(H2S供体)+LPS组、炔丙基甘氨酸[胱硫醚-γ-裂解酶(CSE)抑制剂,PPG]+LPS组、CCK-8+LPS组、PPG+CCK-8+LPS组和CCK-8组。收藏指正
6.The effects of different EAA receptor antogonists on CCK 8 release from ischemic rat striatums.
不同氨基酸受体拮抗剂对缺血纹体CCK-8释放的影响收藏指正
7.2.Proglumide (PGM, CCK antagonist)into PVN significantly blocked the increase of EMG frequency caused by CCK-8 into PVN (P> 0. 05).
2．向PVN先注入CCK拮抗剂丙谷胺（PGM）后注入CCK－8，肌电增加效应被阻断（P＞0．05）；收藏指正
8.The Roles of HO-1/CO in CCK-8 Attenuating Acute Lung Injury Induced by LPS
HO-1/CO在八肽胆囊收缩素减轻内毒素所致的急性肺损伤中的作用收藏指正
9.An increase in DA contents from Acb was found after CCK 8 microinjected into Acb ( P <0 05), but there was no significant change after CCK 8 injected into VTA ( P >0 05).
正常大鼠 Acb微量注射 CCK- 8能使 Acb的 DA含量增高 ( P<0 .0 5) ; 而 VTA微量注射 CCK- 8则不影响 VTA和 Acb区的 DA含量 ( P>0 .0 5)。收藏指正
10.Immunomodulatory Effect of CCK=8 on Reversing Endotoxic Shock and Preliminary Study of Its Molecular Mechanisms
CCK-8抗内毒素休克时的免疫调节作用及其分子机制初探收藏指正
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