dex-induced
2.CONCLUSION: We established DEX-induced mouse thymocyte apoptosis model in vitro, readily for quantitative study.
3.With the stimulation of 0, 25, 50 umol/L PD and combined with 1 umol/L DEX, theapoptosis to necrosis process showed faster trend than DEX group; results showed PDspeeds DEX-induced mouse thymocyte apoptosis.
4.Our data demonstrated that the expression level of GRbeta in TM cells can regulate DEX-induced suppression of phagocytotic activity.
5.Chapter 2 Study on the MAPK related signaling pathway in DEX-inducedmouse thymocyte apoptosis processAIM: To study on the MAPK related signaling pathway in DEX-induced mouse thymocyte apoptosis process and to explore inner mechanism.
6.Dex-induced inunaremppusion could be reversed ther GR was blocked with 25 mg/kg of RU486,when the above immune indexes in chickens chickens with 10 mg ～ 75 mg/kg of Dex for 24 hours after GR blocking were greasy higher than those in chickens treated with aam bine(p < 0. 01 ).
9.2. Immunocytochemical technique and reverse transcription-polymerase chain reaction (RT-PCR) were used to examine the effects of NF-кB specific inhibitor, PDTC and dexamethasone (DEX) on NF-кB activation and ICAM-1 expression induced by CSE in mouse macrophages.
10.It also remarkably enhanced SRBC-induced foodpad DTH reaction in DEX-treated mice.
