dystrophic
1.The condition of being dystrophic.
2.Based on the distincthereditary style. DEB is divided into dominant dystrophic epidermoIysis bullosa(DDEB) and recessive dystrophic epidermolysis bullosa (RDEB).
3.Duchenne muscular dystrophic mice treated by bone marrow stem cells transplantation
4.Postnatal scan of the right dystrophic kidney and the normal left kidney.
5.Effect of DDB on dystrophic hamsters:An in vivo and in vitro study
6.Differential expression of nNOS mRNA and nNOS protein in normal and Duchenne muscular dystrophic muscles
7.Dystrophic calcification of soft tissue or cartilage is the result of calcium deposition in damaged tissues.
8.Dystrophic epidermolysis bullosa (DEB) is caused by mutations in the COL7A1 gene encoding type Ⅶ collagen, the major components of anchoring fibrils.
9.Results: The expressions of CTGF and TGF-β1 were distinctly increased in the dystrophic muscles of children with PMD. CTGF was weakly expressed in the muscle of older children with CMD.
10.Methods We used dystrophin minigene SMCKA3999 to construct rAAVSMCKA3999. When injected into the skeletal muscle of mdx mice(DMD model), we adopted methods of immunofluorescent (IF) staining, Evans Blue and electromicroscopy to observe if rAAVSMCKA3999 could effectively ameliorates dystrophic pathology in mdx model mice.
