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1.Glucocorticoid-induced DNA Fragmentation in CEM-C7 Gells
糖皮质激素对CEM-C7细胞DNA的断裂作用收藏指正
2.Experimental study on Gui Si Bu tablets in preventing glucocorticoid -induced osteoporosis in rats
中药龟丝补骨片防治大鼠类固醇性骨质疏松的实验研究收藏指正
3.A decrease in phagocytic activity has been proposed in the pathogenesis of glaucoma and glucocorticoid-induced glaucoma.
在青光眼和激素性青光眼的发病机制的研究中已经发现小梁网细胞的吞噬功能有所下降。收藏指正
4.The authors investigated whether the beta-adrenoceptor antagonist propranolol blocks glucocorticoid-induced memory retrieval impairments in human subjects.
作者研究了β肾上腺素能受体拮抗剂心得安是否阻断受试人群中糖皮质激素诱导的记忆重现损伤。收藏指正
5.Objective:To discuss the Silvia Miltiorrhiza Concrete Injection and Homogeneous Allograft Bone with Deeply Refrigeneous-Preserved on Treatment of the Glucocorticoid-Induced Avascular Necrosis of Femoral Head (GANFH).
目的:观察丹参液配合同种异体冻干骨移植治疗激素性股骨头坏死的机理。收藏指正
6.Experimental Study on Glucocorticoid-Induced Osteoporosis and the Effects of Water Extract of Danshen (DS) and Its Bioactive Compound Danshensu (DSU) in Vivo and in Vitro
丹参水提取物(DS)和丹参素(DSU)对实验性糖皮质激素性骨质疏松的作用及机制研究收藏指正
7.Material and Methods To construct rabbit glucocorticoid induced glaucoma model by using 1% DEX solution eyedrops and then contrast the differences of expression of AQP-1 and 4 in NPCE and TM with normal controls by using immunohistochemistry and immunofluorescence methods, as well as Western blot method.
材料与方法用1%地塞米松磷酸钠溶液作用于兔眼,建立激素性高眼压模型。 然后通过免疫组化、免疫荧光和western blot方法比较高眼压组和正常对照组兔眼无色素睫状上皮和小梁组织AQP1和AQP4表达水平的差异,分析地塞米松对它们的影响及与眼压的关系。收藏指正
8.Objective To study the relationship between the production of aqueous humor and the tissue-specific expression of AQP1 and AQP4. And also study the effects of dexamethasone (Dex) on the expression of aquaporin-1(AQP1) and aquaporin-4(AQP4) in rabbit non-pigmented epithelium of ciliary body (NPCE) and trabecular meshwork(TM) cells, thereby to probe into the pathogenesis of glucocorticoid induced glaucoma.
目的观察AQP1和AQP4在兔眼组织中的分布与房水分泌的关系。 研究地塞米松对兔眼睫状体无色素上皮及小梁组织内皮细胞AQP1和AQP4表达的影响,探讨激素性青光眼发病机制。收藏指正
9.OBJECTIVE: Elevated glucocorticoid levels impair retrieval of emotional information, and animal studies indicate that this effect depends on concurrent emotional arousal-induced increases in noradrenergic transmission within the brain.
目的∶糖皮质激素水平升高损害了情感性信息的重现,动物研究表明这种作用依赖于脑内去甲肾上腺素能递质传输情绪的唤醒-诱导同时增加。收藏指正
10.Stress-induced activation of the hypothalamus-pituitary-adrenal (HPA) axis and the consequent increase in plasma adrenocorticotropic hormone(ACTH) and glucocorticoid levels are dependent upon CRH.
应激诱导的HPA激活及随后血浆促肾上腺皮质激素(adrenocorticotropic hormone ,ACTH)及糖皮质激素(glucocorticoid ,GC)含量的升高均依赖于CRH。收藏指正
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