induced action
2.The mechanism of inactivation-induced change of action potentials ofmuscle fibres and the possible reason for the difference in the recovery processbetween EDL and SOL are discussed.
5.The changes in rat plasma level of endothelin-ir (ET-ir) during CCl4-induced hepatic necrosis and the therapeutic action of ET-antiserum (ET Ab) were observed.
7.The quinidine-induced EAD could be abolished and the duration of action potential was recovered to normal value either by tetrodontoxin (1.0μmoL/L) or by nifedipine (0.5-1.0 μmol / L).

