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1.Biochemical investigations showed hyponatremia, hyperkalemia, hypochloremia, metabolic acidosis and large amount of urinary sodium excretion.
经过适当治疗和水分补充,临床肠胃症状很快获得缓解。收藏指正
2.Results showed that urinary sodium excretion(sodium intake )were adolescence 143.85 mmol/d,young men 253.7 mmol/d,old men 184.4 mmol/d.
结果显示三组膳食钙的摄入量均低于我国供给量。 2 4 h尿排出量 (即膳食摄入量 ) :青少年组 1 43.85mmol/d,青年组 2 53.7mmol/d,老年组 1 84.4mmol/d,三组间差异显著 ( P<0 .0 0 1 )。收藏指正
3.It was also found that the level of renal sodium excretion decreased and the erythrocyte sodium content increased during the high salt intake stage (?P?<0?05 or 0.01), and 24 hour urinary sodium excretion was negatively correlated with the level of PNE (?r?=-0.54, ?P?<0?05).
盐负荷期间24小时尿Na+排泄量降低,红细胞Na+含量显著升高,且前者与PNE呈负相关(r=-0.54,P<0.05)。收藏指正
4.EFFECT OF INTRACEREBROVENTRICULAR INJECTIONS OF ANG Ⅱ AND ANG Ⅱ ANTIBODY ON RENAL SODIUM EXCRETION AND Na~+·k~+-ATPase IN RAT
大鼠脑室内注射ANGⅡ和ANGⅡ抗体对尿排出和肾皮质Na~+·K~+-ATPase的影响收藏指正
5.24 hour urinary sodium excretion was examined in 25 hyponatremia patients, 34 patients aged over 80 and 31 patients aged 60 ~ 79 without hyponatremia.
同时选择80岁以上非低血症患者(高龄对照组)34例及60-79岁非低血症患者(老年对照组)31例同样测定24 h尿排泄量作为对照。收藏指正
6.Excretion of excessive amounts of sodium in the urine.
尿排泄增加尿中丢失大量的阳离子收藏指正
7.On, the other hand, creatinine excretion, calcium excretion and urinary sodium/potassium ratio increased with age.
反此尿钙及肌酸肝排泄量随年龄而增加。收藏指正
8.Potassium is in competition with hydrogen ion for renal tubular excretion in exchange for sodium ion.
钾离子和氢离子在肾小管排泄中相互竞争和离子交换。收藏指正
9.METHODS Acute liver damage model was established with carbon tetrachloride (CCl_4) administration to male Sprague-Daw- ley rats. Twenty-four and 48 hours later after CCl_4 administration, the excretion of acute sodium and water load was examined,and 24 hours later after normal saline administration,the excretion of acute sodium and water load was examined in control group.
方法有四氯化碳对 SD 大鼠复制急性肝损伤模型,用四氯化碳后24 h,48 h 做急性水负荷排泄实验,对照组注射等量生理盐水,24 h 后做急性水负荷排泄实验.收藏指正
10.Thus, in alkalosis, the increased potassium ion excretion in exchange for sodium ion permits hydrogen ion conservation.
因此在碱中毒中,钾交换的增加使氢离子保留。收藏指正
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